*sigh*
not this again
You'd do better to expand upon why you think he's incorrect. What's wrong with a caloric deficit based approach?
*sigh*
not this again
LOL
You've probably never even heard of ghrelin, leptin, incretin, glucagon, neurotensin and various other neuropeptides. You probably don't know what the POMC neuron is.
LOL
You'd do better to expand upon why you think he's incorrect. What's wrong with a caloric deficit based approach?
So someone can burn 2000 calories a day and eat 1000 calories a day and gain weight then?
False. Insulin does not regulate sugar levels. It facilitates in sending glucose to various parts of your body from the blood stream to a cellular level. It is used not just as a source of energy feeding your skeletal muscle tissue, your brain, etc - but also aids in recovery of said tissue during damage.
It does not regulate "levels". It helps your body absorb the glucose.
Also, sugar isn't the culprit - it is GLUCOSE. Glucose is what carbohydrates are turned into when your body digests them. Carbs as in bread, pasta, whole grains, wheat, sugars, etc.
They are all glucose.
Eating large amounts of whole grains produces the same effect as eating large amounts of sugar - the main difference is how quickly your body can turn a raw ingredient such as whole grains into glucose vs a refined material like table sugar.
Planting the blame solely on "sugar" foods like candy, snacks, pop, etc is asinine and misleading. No shit high amounts of glucose are dangerous - why not blame ALL foods high in carbohydrates and not just "sugar"?
Glucose did not make that happen. Working out did not make that happen. The auto-immune disease did.
Is there a relation between eating large amounts of carbohydrate-heavy foods and diabetes? For type 2, sure. It's insulin resistance. You produce so much of it to help facilitate the carbs you intake that your body begins to become resistant to the hormone - meaning you'll wind up on Metformin and other diabetic meds.
Moderation. That's it. I am a diabetic due to an auto immune disease and I find blaming sugar for so much is flat-out wrong. Pop, candy, snacks are not the only means we get carbs.
Moderate the whole fucking thing - this bullshit about "omg that sugary goodness is bad for you" is just one TINY fucking aspect to a much larger picture.
If these doctors and scientists who come out with these studies REALLY cared - they'd finish out the mile they started instead of stopping after checking out the first 20 yards.
There's so much more to the glucose picture than just "sugar". Demonizing one part does nothing to help curb the problem when so many equally dangerous parts exist.
Also - do we REALLY need a study to tell us "eating too much of a bad thing is bad for you"? REALLY? No fucking shit! Thanks for the tip! Nobody knows this!
The better effort would be to help people understand how to properly moderate their food intake - but no - we get continuous research into shit everyone already knows.
Sugar isn't bad in moderation. An alcoholic drink here and there won't turn your liver toxic. An over-abundance, will. Same shit here.
Instead of demonizing something that can be A-OK in moderation - how about EDUCATING on how to properly moderate.
EDIT:
Also saying that foods will affect everyone the same is stupid, as well. As is proven time and again what works for A might not work for B.
Pretty much nobody succeeds long term just counting calories. The long term trials on calorie restriction are absolutely dismal. The neuroendocrine system will always trump willpower in the long run. However, you can do things that affect the neuroendocrine system to reach a lower set point so that you will naturally eat less calories and lose body fat.
Yeah, I don't disagree there with the psychology and the difficulty...but if we're just talking pure numbers, how does one gain weight if they are on a caloric deficit? Someone mentioned burning off 2000 for every 1000; putting aside the actual number estimates, I'm curious to your reasoning of how someone's body can gain weight from negative calories.
LOL
You've probably never even heard of ghrelin, leptin, incretin, glucagon, neurotensin and various other neuropeptides. You probably don't know what the POMC neuron is.
LOL
They all trigger insulin responses. The strength of which varies. Insulin is the hormone responsible for regulating fat cells.
You'd do better to expand upon why you think he's incorrect. What's wrong with a caloric deficit based approach?
All those things you have listed are affected by your caloric intake (as well as other factors). Over a long period of time where you restrict your calories, the rate of leptin production, for example, decrease and thus your metabolic rate falls, making it more difficult for you to lose weight after a while by merely restricting your caloric intake but there is a net weight loss. A person has to pick up the slack and exercise as well to increase the metabolic rate.
Ghrelin secretion has little to do with caloric intake and more to do with how much "filled up" your stomach is. That is why ghrelin levels have been seen to be significantly reduced in people who have smaller stomachs, either due to surgery or naturally.
Glucagon is directly related to blood sugar levels, though, and something that actually supports the guy's theory about calories. The less you eat, the lower your blood sugar, the more glucagon induces gluconeogenesis and the more release of glucose from cells. This eventually leads to weight loss since your fat stores have to be broken down to increase blood sugar levels.
The truth is, both hunger and caloric intake are what come into play when talking about weight gain and loss. If you restrict your caloric intake for a substantial amount of time, your body's homeostatic state is affected and thus there is a change in set points. A person would feel satiated with a lesser amount of food if he eats fewer calories over a long period of time.
This is a similar effect as type 2 diabetes, where your body develops resistance to insulin. In this case, your body becomes more resistant to the hunger-inducing hormones or may produce lesser and lesser amounts as it gradually adjusts to the decreased caloric intake.
It's true that lowering calories can change the neuroendocrine system and allow people to keep weight off, in some people, but it's still a pretty dismal method of keeping weight off. Just this week, the look AHEAD trials was halted 11 years in (it was supposed to be 13.5 years long) because it was obvious that calorie restriction (and other lifestyle interventions -- i.e. exercise) wasn't going to make a difference. Weight loss was minimal and the difference in CVD events was going to be non-significant.
I mentioned the various hormones/peptides because they aren't exactly tied to calorie intake. Ghrelin is also affected by quality of sleep. People who get inadequate amounts of sleep or have a sleep disorder like sleep apnea have higher levels of ghrelin which leads to a greater appetite. Ghrelin is also not just affected by energy density. A meal consisting primarily of fat and protein will suppress ghrelin much longer than a meal consisting primarily of carbohydrate. In fact, carbohydrate strongly suppresses ghrelin initially, but there is a rebound effect ~3 hours later where ghrelin rises above baseline. Protein strongly suppresses ghrelin, without the rebound effect and fat minimally suppresses ghrelin, but does so for a while and without the rebound effect.
I'm not sure about the set point changing just by eating at a calorie deficit for a while. Most people re-gain their weight after calorie restriction. If you take a weight reduced person and match them to someone else who is the same weight but not weight reduced, the person who is weight reduced will have less leptin than the non weight reduced person. Leptin also controls hunger. More leptin = less hunger and higher metabolic rate, thus the weight reduced person will struggle to maintain his/her reduced weight compared to the non weight reduced person. Interesting, overweight people tend to have high levels of circulating leptin, but it isn't acting in the brain (leptin resistance), likely due to elevated triglycerides. So giving an obese person leptin won't reduce their weight, but giving a weight reduced person allows them to much more easily maintain their weight loss.
Or how about you worry about yourself and allow a free country to remain that way.
We need education, not regulation.
Paleo diet. Sugar is extremely limited to just fruit or none at all. I am safe here .
How many do you eat? I eat (no joke) an apple a day at least 6 times a week.
HFCS is in everything.
Not sure why the US is so obsessed with using HFCS in all their foods instead of cane sugar.
I never said that someone can gain weight with a negative calorie deficit.
storafötter;43505631 said:I cant believe that people are trying to compare grains to sugar. Next is how dangerous it is to eat fruit due to the sugars in fruit!. Nevertheless I find this new science interesting. I remember reading also about how sugar disrupts your hormones. There has never been any good benefits for eating refined sugar. I try to limit my sugar intake but I used to be better at it before.
You'd do better to expand upon why you think he's incorrect. What's wrong with a caloric deficit based approach?
C'mon man, that stuff is highly toxic, you can't be serious with this argument.By that logic, why not put arsenic in food? Or all kinds of carcinogenic substances?
I'm against more regulations in most cases, but the anything concerning health should be regulated strictly.
C'mon man, that stuff is highly toxic, you can't be serious with this argument.
Nope.Jack_AG said:Carbs as in bread, pasta, whole grains, wheat, sugars, etc.
They are all glucose.
Eating large amounts of whole grains produces the same effect as eating large amounts of sugar - the main difference is how quickly your body can turn a raw ingredient such as whole grains into glucose vs a refined material like table sugar.
Just in time for Halloween!
I can't believe this discussion missed the most obvious example of why this reasoning is bullshit: Asian diets.
Asian (including Indian subcontinent) diets are traditionally very high in carbs in the form of rice, consumables made from rice based flours (noodles, buns, etc), fruits, and vegetables. And yet until Western eating habits started to influence Asian countries, Asians generally suffered very low rates of obesity, low rates of diabetes, and low rates of various diet-linked ailments like heart disease. Both of which have been on the rise due to increased consumption of diets richer in animal based fats (excluding fish) and refined sugars.
Those of you who have young kids should try an experiment: cut as much sugar out of your child's diet as possible for a week and monitor behavioral changes. This is easier with young kids because you can have absolute control over all of the variables.
I have an 18mo and a few weeks back, we noticed she was suddenly becoming whiny, not listening to us, and breaking her normal sleep patterns. Just the terrible-twos? On a hunch, I told my wife to go through her snacks with me and found one "fruit-bar" where the second ingredient was sugar.
Sure enough, we took it out of her diet and it was like night and day. With no other change in her diet, cutting out one snack -- a "fruit bar" -- where she was getting probably over 50% of her daily sugar corrected her behavioral flare ups. We replaced it with another, much funkier tasting fruit bar where the first ingredient is rolled oats and 4/8 ingredients are dried fruits with no additional sugar --
We've generally kept her on a very sugar restricted diet but not carb restricted. She still gets things like grilled cheese sandwiches, pastas, rice, low-sugar cereals, and so on, but cutting out the sugar made a big difference in behavior and sleep patterns.
I'm glad her behavior problems went away, but this seems to be post hoc reasoning. You changed one factor of her diet, but did you control for anything else? Social interactions, etc? Did you ever punish her for her whiny behavior, or praise her when she was well behaved during that period?
Jack_AG said:Carbs as in bread, pasta, whole grains, wheat, sugars, etc.
They are all glucose.
Not obsessed. The populace barely cares. It's all big business, corn subsidies and contracts going back decades.
Let me guess.. is it cheaper?
Toronto Star
Bad news sugar lovers: a diet high in fructose wont just make you fat, it may also make you stupid, according to research out of California.
A steady high-fructose diet disrupts the brains cognitive abilities, leading to poor learning and memory retention, says a study by Fernando Gomez-Pinilla, a neurosurgery professor at the David Geffen School of Medicine at UCLA and Rahul Agrawal, a visiting UCLA postdoctoral fellow from India.
This type of diet. . . (affects) the transmission of information across cells. . . learning and memory and practically any type of brain function depends very much on how transmission is transported across cells, Gomez-Pinilla said in an interview with the Star.
Health concerns
Their study, published in the May 15 edition of the Journal of Physiology, looked at high sugar consumption, focusing less on naturally occurring fructose in fruits and more on the fructose in high-fructose corn syrup.
Research has already proven a high-fructose diet leads to a slew of health concerns, including obesity, diabetes and fatty liver.
The U.S. is the worlds largest consumer of sweeteners.
High-fructose corn syrup, which acts as a preservative and sweetener, is found in a variety of processed foods, from soft drinks and baby food to salad dressings and condiments.
The average American consumes approximately 21 kilograms of cane sugar and 16 kilograms of high-fructose corn syrup annually, according to the U.S. Department of Agriculture.
Role of fatty acids
Gomez-Pinilla and Agrawal studied two groups of rats, both of which drank a fructose solution in their drinking water for six weeks. One of the groups also consumed omega-3 fatty acids, from flaxseed oil and a DHA (docosahexaenoic acid) capsule. Omega-3 fatty acids have been found to guard against heat disease, high cholesterol and mental conditions such as bipolar disorder and depression, according to the University of Maryland Medical Center.
Both rat groups were trained on a maze for five days before starting their new diet. After six weeks, Gomez-Pinilla and Agrawal retested the rats on the maze to monitor brain function and memory retention, noting the rats that consumed the fructose solution without the omega-3 fatty acids had problems with how they were able to think and recall routes in the maze.
Those rats also showed a resistance to insulin, a hormone that regulates sugar levels in the body.
Rats fed on a (omega-3 fatty acids) deficient diet showed memory deficits in a Barnes maze, which were further exacerbated by fructose take, the authors write.
They found that a rich diet of omega-3 fatty acids counteracted the negative affects of fructose.
Implications for humans
In terms of humans, Gomez-Pinilla predicts such changes in the brain to happen within six months to a year.
The implication(s) here (are) the high consumption and the chronic consumption for man, Gomez-Pinilla said, adding research needs to be done on the specific affects on humans.
We dont know yet how long (the damage) can last.
The war on unhealthy food choices is a growing. In September, New York City announced it would ban sugar-filled drinks larger than 16-ounces from concession and fast-food stands, restaurants and movie theatres.
In Canada, a push on healthy eating is on the rise as the country grapples with the fact that 31.5 per cent of Canadian children aged 5 to 17 are either overweight or obese, according to a Statistics Canada report released in September.
Diabetes is very prevalent in western society. Its known already there is an (epidemic) of diabetes, which is highly related to a consumption of foods high in sugar, Gomez-Pinilla said.
Gomez-Pinilla advocates a nutrient-rich diet that includes omega-3 fatty acids and a proper mix of healthy choices to offset the dangers of fructose.
Foods that are rich in omega-3 fatty acids include flaxseed oil, some types of fish, such as salmon, and nuts.
Stopped drinking soft drinks long, long time ago so I am good ^_^
Those of you who have young kids should try an experiment: cut as much sugar out of your child's diet as possible for a week and monitor behavioral changes. This is easier with young kids because you can have absolute control over all of the variables.
I have an 18mo and a few weeks back, we noticed she was suddenly becoming whiny, not listening to us, and breaking her normal sleep patterns. Just the terrible-twos? On a hunch, I told my wife to go through her snacks with me and found one "fruit-bar" where the second ingredient was sugar.
Sure enough, we took it out of her diet and it was like night and day. With no other change in her diet, cutting out one snack -- a "fruit bar" -- where she was getting probably over 50% of her daily sugar corrected her behavioral flare ups. We replaced it with another, much funkier tasting fruit bar where the first ingredient is rolled oats and 4/8 ingredients are dried fruits with no additional sugar -- she still seems to like it, luckily. (And by the way, the ability for me to easily look at and compare the ingredients and nutritional information is the result of government regulation on food labeling -- a perfect example of how government regulation can work without taking the form of an onerous ban)
We've generally kept her on a very sugar restricted diet but not carb restricted. She still gets things like grilled cheese sandwiches, pastas, rice, low-sugar cereals, and so on, but cutting out the sugar made a big difference in behavior and sleep patterns.
I'll just stick to diet sodas then, and other products containing sweeteners.
There's actually some debate about weather these cause spikes as well. Basically, your body is like, "OH SHIT INCOMING SUGAR" when there isn't any.